Gingival hyperplasia is excessive growth or thickening of gum tissue. The phase IV clinical study analyzes which people take Trileptal and have Gingival hyperplasia. GO is characterized by the accumulation of extracellular matrix in gingival connective tissues, particularly collagenous components, with varying degrees of inflammation. Specialty Gastroenterology Gingival enlargement is an increase in the size of the gingiva (gums). Primary hyperplastic gingival hyperplasia: Primary low inflammatory, rather gelatinous swelling of the marginal and interdental gingiva, especially in the frontal parts. Surgical therapy was carried out to provide a good . Gingival hyperplasia is found among people who take Furosemide, especially for people who are male, 60+ old, have been taking the drug for 1 - 6 months. Gingival hyperplasia, also referred to as gingival enlargement, is the overgrowth of gum tissue around your teeth. The complex of local and systemic factors which determines the variations in gingival inflammatory response to injury is only partially known. There are several drugs that have been associated with gingival overgrowth (the increase in matrix protein form of enlargement) including phenytoin The most common is chronic inflammatory gingival enlargement, when the gingiva presents clinically as soft and discolored. Gingival enlargement can be caused by numerous factors which may include inflammatory conditions or be the side effect of specific medications. In chronic or severe cases, inflammation and its secondary effects (mineral or calcium deposition) may be observed. Inflammatory enlargements are characterized by swelling or edema, redness, and a tendency to bleed during tissue manipulation ( Figures 14-9 and 14-10 ). Definition / general. Drug-induced gingival hyperplasia (DIGH) causes problems with chewing, aesthetics, and pronunciation, and leads to the deterioration of the patient's quality of life (QOL). Purpose: Gingival enlargement, also synonymous with the terms gingival hyperplasia or hypertrophy, is defined as an abnormal overgrowth of gingival tissues. Causes include: Hormonal states: pregnancy, puberty; Nutritional deficiency: scurvy (vitamin C deficiency); Medications, most often ciclosporin . Hyperplasia specifically means an increase in the number of cells in the gum tissue. Bad breath. There are a number of causes for this . There are several causes of gingival enlargement and they can be grouped into four categories: 1) inflammatory gingival enlargement, 2) medication-induced gingival enlargement, 3) hereditary . Familial or hereditary conditions: examples include familial fibromatosis, idiopathic fibromatosis, gingivomatosis, and hereditary gingival hyperplasia. Infection and pus formation. IDIOPATHIC GINGIVAL ENLARGEMENT termed as gingivostomatitis, elephantiasis, idiopathicfibromatosis, hereditary gingival hyperplasia & congenital familial fibromatosis. Systemic-induced - conditions such as leukemia, pregnancy and hormonal imbalances help in promoting gingival hyperplasia. . Gingival hyperplasia is a term used to describe the abnormal growth of excessive gum tissue. The visible signs and symptoms of gingival hyperplasia include -. Frequently occurring as a side effect of medication, more rarely in neurological . One of the main drugs associated with GO is the antiepileptic . Gingival hyperplasia is found among people who take Trileptal, especially for people who are female, 20-29 old. Other symptoms can include pain, swelling, and bleeding of the gums. Also called fibrous hyperplasia. Treating the underlying cause of gingival hyperplasia can help resolve the condition. It is a common feature of gingival disease. Clinically, the average age of patients in this study was 11.8 years, with a 2.3:1 female predominance. This is caused by tissue edema and infective cellular infiltration caused by prolonged exposure to bacterial plaque, and is treated with conventional periodontal treatment, such as scaling and root planing. Gingival hyperplasia is an overgrowth of gum tissue around the teeth. Occasionally, chronic inflammatory gingival enlargement occurs as a discrete sessile or pedunculated mass that resembles a tumor. Gingival enlargement, the currently accepted terminology for an increase in the size of the gingiva, is a common feature of gingival disease. 40, 41. Gingival enlargement can be induced by three main causes stemming from: Inflammation, Medication, and Systemic disease Local and systemic factors influence the gingival conditions of the patient. Bad breath. It can be associated with chronic periodontitis. Treatment can be in the form of natural healing, once the etiology is removed, by instituting proper dental hygiene, and through nonsurgical treatment and/or surgical treatment. 1 Because gingival hyperplasia denotes a specific histologic diagnosis, clinicians must rule out other causes of gingival enlargement with biopsies of affected tissues. Gingival hyperplasia is an overgrowth of gum tissue around the teeth. Local and systemic factors influence the gingival conditions of the patient. 5) False enlargement. A case of a 19-year-old male presenting with maxillary and mandibular chronic inflammatory gingival enlargement associated with prolonged orthodontic therapy is reported here. Oral mucosa being constantly subjected to various stimuli manifests with a spectrum of disease which may be developmental, inflammatory, reactive or neoplastic [].These lesions may manifest as either generalized or localized [1, 2].Fibro-epithelial hyperplasia, a histological variant of fibroma is a proliferative fibrous lesion of the gingival tissue that hinders function and aesthetics. Gingival hyperplasia represents an excessive gum growth. Various Types of Inflammatory Gingival Enlargement A. Mesodermal Hyperplasia. The enlargement of the gingival tissue may occur due to many other reasons also. Swelling of the gums. These factors results in a spectrum of diseases that can be developmental, reactive and inflammatory to neoplastic. Abstract We report 52 cases of a unique and pathologically distinctive form of inflammatory gingival hyperplasia in young patients. We have identified gingival tissue-specific mesenchymal stem cells (GMSCs) and found their functional change being correlated with drug-induced gingival hyperplasia. Gingival hyperplasia is found among people who take Depakote, especially for people who are female, 50-59 old, have been taking the drug for < 1 month. Gingival overgrowth, hypertrophic gingivitis, gingival hyperplasia, or gingival hypertrophy is the other names of . Gingival nodules may represent one of the following: Reactive/inflammatory gingival nodules, solitary or diffuse; these are by far the most common Peripheral (extraosseous) odontogenic cysts and tumors (see Chapters 14 and 15 ) Soft tissue tumors (such as nerve sheath or smooth muscle tumors discussed in Chapter 6 ) The lesion is mostly asymptomatic and color of the . The fibrotic hyperplastic gingival tissues will, to a large extent, persist even following elimination of the initial source of irritation, and those anatomical changes may predispose to future irritation. . In the past, the terms gingival hyperplasia and gingival hypertrophy have been used to describe this condition. Abstract Background information: Human gingival tissues are prone to hyperplasia under inflammatory stimuli. Associated with diphenylhydantoin (Dilantin) ingestion; also cyclosporine A, nifedipine and other drugs. Gingival hyperplasia can be caused by inflammatory conditions and the side effects of certain medications.. . These are inflammatory response to local irritant . Gingival hyperplasia is found among people who take Lipitor, especially for people who are female, 60+ old. Local and systemic factors influence the gingival conditions of the patient. Gingival Hyperplasia/hypertrophy main causes are inflammatory, drug-induced, or systemic Clinical Features Inflammatory Gingival Hyperplasia Gingival Hyperplasia from Diltiazem Swollen gums Bright red or purple +/- bleeding and easily bleed with probing Poor oral hygiene Differential Diagnosis Inflammatory hyperplasia Gingivitis The treatment is based on the cause. In acute reactive gingival hyperplasia the gums are red, boggy, and bleed easily with minor trauma. The edema caused by inflammation causes the gums to swell, while the activated inflammatory mediators lead to overgrowth of the gums. The phase IV clinical study analyzes which people take Furosemide and have Gingival hyperplasia. The inflammation is often caused by plaque buildup on the teeth from food, bacteria, and poor hygiene practices. Classification. It may be soft, which bleeds easily, or firm and fibrous. The inflammation can make the gums tender and red, and it can trigger bleeding. Definitions Gingival Hyperplasia Non-inflammatory thickening of gum tissue (via increased cell number) overlying the alveolar ridge Gingival Hypertrophy Abnormal gum enlargement (via swelling of existing cells) III. Gingival hyperplasia can happen due to various causes, including plaque buildup or oral inflammation, side effects from medication, and systemic conditions such as Crohn's disease. It is also more likely to occur in those with poor dental hygiene. This increase in gingival cells has many different causes, but one of the most common is drug-induced gingival hyperplasia. This condition is more common in Boxers, Great Danes, Collies, Mastiffs, and Retriever breeds but may be seen in any breed. There are a number of causes for this condition, but it's often a symptom of poor oral hygiene or a side effect of using. Definition. Rather than getting gingival recession, they get hyperplasia. Gingival hyperplasia typically refers to the increase in the number of cells, whereas gingival hypertrophy deals with the increase in cell size. Gingival hyperplasia Occasionally, plaque buildup or gum irritation caused by braces may trigger a condition known as gingival hyperplasia . Gingival hyperplasia can occur as a direct result of inflammation. Pathophysiology Normal pathway of Collagen degradation is disrupted, allowing for increased extracellular Collagen deposition at gums Gingival Enlargement. In our study, fragments of gingival tissue of healthy volunteers were collected during operation. 1 ), such as drugs (anticonvulsants, cyclosporine, calcium channel blockers, erythromycin), infections and autoimmune hormonal changes (pregnancy, puberty), nutritional deficiencies (vitamin C, scurvy . It is created by eHealthMe based on reports of 251,399 people who have side effects when taking . Presence of fluid may give them a bluish hue and they may lead to resorption of the labial bone due to pressure. Gingival enlargement, the currently accepted terminology for an increase in the size of the gingiva, is a common feature of gingival disease. inflammatory cytokines [30, 33,34,35,36], and 3 . The gummy smile is caused by gingival enlargement and can be induced by three main causes: (1) inflammation, (2) medication, and (3) systemic disease. Profile . The role of lymphangiogenesis during development of periodontitis is unknown. Chronic inflammatory gingival enlargement originates as a slight ballooning of the interdental papilla and/or the marginal gingiva. Gingival hyperplasia, also known as gingival enlargement, is a swelling of the gum tissue (gingiva). This swelling is often generalized, affecting all of the visible gum tissue within the mouth. . Inflammatory gingival enlargement. However, it can also appear in patients with good oral hygiene, so in these cases, other factors could cause local irritation. The present case report describes a case of long standing gingival enlargement in a systemically Buildup of plaque on the teeth. Orofacial granulomatosis (OFG) is a rare inflammatory condition which causes persistent or recurrent enlargement of the oral and maxillary soft tissues including the gingivae ( Fig. Increased vulnerability of the gingiva can lead to secondary inflammatory superimposition. as soc iat ed wit h gin giv a. . Vascular endothelial growth factor C (VEGFC) is the main known lymphangiogenic growth factor, and its expression is increased in periodontitis, a common chronic infectious disease that results in tissue destruction and alveolar bone loss. Gingival enlargement can be caused by a number of factors, including inflammatory conditions and the side effects of certain medications. Home Oral cavity & oropharynx > Gingival hyperplasia. ETIOLOGY : unknown hereditary basis (autosomal dominant or recessive) begins with primary & secondary dentition eruption. Gene expression of 29 genes was investigated in gingival fibroblasts cell culture treated with gabapentin, compared with untreated cells. The gingiva is pink, non-tender, involves the attached gingiva, the gingival . Specifically, the presence of plaque is usually the most common etiologic factor. Whereas some people have too little gums to cover their teeth, those with this condition have too much gum tissue. The treatment for the condition is determined based on the cause. It is created by eHealthMe based on reports of 20,140 people who have side effects when taking Trileptal from the FDA, and is updated . leads us to think that gabapentin can interact and cause post-inflammatory gingival hyperplasia, but . 11 ). Gingival enlargement is an increase in the size or inflammation of the gingiva. unusu al fibro tic gingival enlarge ment of local ized . These microscopic distinctions are both indicative of a disease process. May require surgical excision. It presents as . Gingival hyperplasia is most commonly observed in Boxer Dogs. Gingival hyperplasia is a condition that refers to an overgrowth of your gums (also known as your gingiva). Gum tissues are mostly tender, red, soft and tend to bleed easily. The phase IV clinical study analyzes which people take Lipitor and have Gingival hyperplasia. . [6] Gingival hyperplasia Gingival hyperplasia ( Table 9.3) may result from local inflammation or gingivitis and infiltrative processes that provide a clue to underlying systemic disease. form of enlargement is inflammatory hyperplasia which is due to plaque induced inflammation of the gingival tissues. Gingival hyperplasia is defined as an enlargement of the gingiva that is noninflammatory, produced by factors other than local irritation, and the result of an increased number of cells.

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